Search results for "Neuronal Plasticity"

showing 10 items of 166 documents

Positive Controls in Adults and Children Support That Very Few, If Any, New Neurons Are Born in the Adult Human Hippocampus.

2020

Adult hippocampal neurogenesis was originally discovered in rodents. Subsequent studies identified the adult neural stem cells and found important links between adult neurogenesis and plasticity, behavior, and disease. However, whether new neurons are produced in the human dentate gyrus (DG) during healthy aging is still debated. We and others readily observe proliferating neural progenitors in the infant hippocampus near immature cells expressing doublecortin (DCX), but the number of such cells decreases in children and few, if any, are present in adults. Recent investigations using dual antigen retrieval find many cells stained by DCX antibodies in adult human DG. This has been interprete…

0301 basic medicineAdultAging1.1 Normal biological development and functioningNeurogenesisHippocampusneural progenitorsHippocampal formationRegenerative Medicinehuman hippocampusMedical and Health SciencesHippocampus03 medical and health sciences0302 clinical medicinedoublecortinStem Cell Research - Nonembryonic - HumanUnderpinning researchmedicineHumansdentate gyrusChildnew neuronsPediatricNeuronsNeurology & NeurosurgeryNeuronal PlasticitybiologyGeneral NeuroscienceDentate gyrusNeurogenesisPsychology and Cognitive SciencesNeurosciencesCell DifferentiationDual PerspectivesHuman brainStem Cell ResearchNeural stem cellDoublecortin030104 developmental biologymedicine.anatomical_structureNeurologicalbiology.proteinStem Cell Research - Nonembryonic - Non-HumanMental healthNeuronNeuroscience030217 neurology & neurosurgeryThe Journal of neuroscience : the official journal of the Society for Neuroscience
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Heat shock protein (Hsp) regulation by muscarinic acetylcholine receptor (mAChR) activation in the rat hippocampus.

2018

The cholinergic system plays a crucial role in modulating in the central nervous system physiological responses such as neurogenesis, neuronal differentiation, synaptic plasticity, and neuroprotection. In a recent study, we showed that Oxotremorine-M, a non-selective muscarinic acetylcholine receptor agonist, is able to transactivate the fibroblast growth factor receptor and to produce a significant increase in the hippocampal primary neurite outgrowth. In the present study we aimed to explore in the rat hippocampus the possible effect of acute or chronic treatment with Oxotremorine-M on some heat shock proteins (Hsp60, Hsp70, Hsp90) and on activation of related transcription factor heat sh…

0301 basic medicineAgonistMalemedicine.medical_specialtymedicine.drug_classPhysiologyClinical BiochemistryNeuronal OutgrowthScopolamineheat shock proteinHsp90NeuroprotectionHippocampusHsp7003 medical and health sciencesmuscarinic receptor0302 clinical medicineHeat Shock Transcription FactorsHeat shock proteinInternal medicineMuscarinic acetylcholine receptormedicineOxotremorineAnimalsRats WistarHSF1Heat-Shock ProteinsNeuronsNeuronal PlasticityChemistryOxotremorineNeurodegenerative DiseasesCell BiologyReceptors Fibroblast Growth FactorReceptors MuscarinicHsp70Rats030104 developmental biologyEndocrinologyheat shock factor 1HSP60030217 neurology & neurosurgerymedicine.drugSignal TransductionJournal of cellular physiology
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Intra-neuronal Competition for Synaptic Partners Conserves the Amount of Dendritic Building Material

2017

Brain development requires correct targeting of multiple thousand synaptic terminals onto staggeringly complex dendritic arbors. The mechanisms by which input synapse numbers are matched to dendrite size, and by which synaptic inputs from different transmitter systems are correctly partitioned onto a postsynaptic arbor, are incompletely understood. By combining quantitative neuroanatomy with targeted genetic manipulation of synaptic input to an identified Drosophila neuron, we show that synaptic inputs of two different transmitter classes locally direct dendrite growth in a competitive manner. During development, the relative amounts of GABAergic and cholinergic synaptic drive shift dendrit…

0301 basic medicineDendritic spinePresynaptic TerminalsBiologyReceptors NicotinicArticleSynapse03 medical and health sciencesDendrite (crystal)Calcium Channels T-Type0302 clinical medicinePostsynaptic potentialSynaptic augmentationmedicineAnimalsDrosophila ProteinsCalcium Signalinggamma-Aminobutyric AcidNeuronsNeuronal PlasticityGeneral NeuroscienceDendritesReceptors GABA-AAcetylcholine030104 developmental biologySynaptic fatiguemedicine.anatomical_structurenervous systemSynaptic plasticitySynapsesDrosophilaNeuronNeuroscience030217 neurology & neurosurgery
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Boosting Action Observation and Motor Imagery to Promote Plasticity and Learning

2018

Neural Plasticity, 2018

0301 basic medicineImagery PsychotherapyBoosting (machine learning)Article SubjectComputer scienceMovementMachine learningcomputer.software_genrestimulationlcsh:RC321-57103 medical and health sciences0302 clinical medicineMotor imageryHumansLearninglcsh:Neurosciences. Biological psychiatry. NeuropsychiatryComputingMilieux_MISCELLANEOUSNeuronal Plasticitybusiness.industryBraincortexEditorial030104 developmental biologyNeurologyAction observationImagination[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Neurology (clinical)Artificial intelligencebusinesscomputer030217 neurology & neurosurgery
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Reduced interneuronal dendritic arborization in CA1 but not in CA3 region of mice subjected to chronic mild stress

2016

Abstract Introduction Chronic stress induces dendritic atrophy and decreases spine density in excitatory hippocampal neurons, although there is also ample evidence indicating that the GABAergic system is altered in the hippocampus after this aversive experience. Chronic stress causes dendritic remodeling both in excitatory neurons and interneurons in the medial prefrontal cortex and the amygdala. Methods In order to know whether it also has an impact on the structure and neurotransmission of hippocampal interneurons, we have analyzed the dendritic arborization, spine density, and the expression of markers of inhibitory synapses and plasticity in the hippocampus of mice submitted to 21 days …

0301 basic medicineMaleDendritic spineDendritic SpinesHippocampusPSA‐NCAMCell CountNeural Cell Adhesion Molecule L1Hippocampal formationBiologyNeurotransmissionAmygdalaHippocampus03 medical and health sciencesBehavioral NeuroscienceMice0302 clinical medicineInterneuronsNeuroplasticitymedicineAnimalsChronic stressCA1 Region HippocampalOriginal ResearchInhibitionNeuronal PlasticityGlutamate Decarboxylasemusculoskeletal neural and ocular physiologyfungiCA3 Region Hippocampalstructural plasticity030104 developmental biologymedicine.anatomical_structurenervous systemExcitatory postsynaptic potentialGAD67Sialic AcidsNeuroscience030217 neurology & neurosurgeryStress PsychologicalBrain and Behavior
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Functional hypoxia drives neuroplasticity and neurogenesis via brain erythropoietin.

2020

Erythropoietin (EPO), named after its role in hematopoiesis, is also expressed in mammalian brain. In clinical settings, recombinant EPO treatment has revealed a remarkable improvement of cognition, but underlying mechanisms have remained obscure. Here, we show with a novel line of reporter mice that cognitive challenge induces local/endogenous hypoxia in hippocampal pyramidal neurons, hence enhancing expression of EPO and EPO receptor (EPOR). High-dose EPO administration, amplifying auto/paracrine EPO/EPOR signaling, prompts the emergence of new CA1 neurons and enhanced dendritic spine densities. Single-cell sequencing reveals rapid increase in newly differentiating neurons. Importantly, i…

0301 basic medicineMaleDendritic spineGeneral Physics and AstronomyHippocampal formationVARIANTSADULT NEUROGENESIS0302 clinical medicineCognitionhemic and lymphatic diseasesReceptors ErythropoietinHypoxialcsh:ScienceNEURONSMultidisciplinaryNeuronal PlasticityPyramidal CellsNeurogenesisQBrainCell DifferentiationHEMATOPOIETIC PROGENITOR CELLSFemalemedicine.symptomProto-Oncogene Proteins c-fosmedicine.drugEXPRESSIONScienceDendritic SpinesNeurogenesisModels NeurologicalBiologyMotor ActivityGeneral Biochemistry Genetics and Molecular BiologyArticle03 medical and health sciencesParacrine signallingPhysical Conditioning AnimalNeuroplasticitymedicineAnimalsHumansErythropoietinMEMORYCognitive neuroscienceGeneral ChemistryHypoxia (medical)RECOMBINANT-HUMAN-ERYTHROPOIETINCellular neuroscienceErythropoietin receptorMice Inbred C57BLMICE030104 developmental biologyErythropoietinPhysical EnduranceIDENTITYlcsh:QTranscriptomeNeuroscience030217 neurology & neurosurgeryGene Deletion
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Immature excitatory neurons develop during adolescence in the human amygdala.

2019

The human amygdala grows during childhood, and its abnormal development is linked to mood disorders. The primate amygdala contains a large population of immature neurons in the paralaminar nuclei (PL), suggesting protracted development and possibly neurogenesis. Here we studied human PL development from embryonic stages to adulthood. The PL develops next to the caudal ganglionic eminence, which generates inhibitory interneurons, yet most PL neurons express excitatory markers. In children, most PL cells are immature (DCX+PSA-NCAM+), and during adolescence many transition into mature (TBR1+VGLUT2+) neurons. Immature PL neurons persist into old age, yet local progenitor proliferation sharply d…

0301 basic medicineMaleGeneral Physics and AstronomyHippocampus02 engineering and technologyAdult neurogenesisHippocampusNeural Stem Cellslcsh:ScienceChildPediatricNeuronsMultidisciplinaryNeuronal PlasticitybiologyBasolateral Nuclear ComplexQNeurogenesisMiddle Aged021001 nanoscience & nanotechnologyMental Healthmedicine.anatomical_structureChild PreschoolExcitatory postsynaptic potentialSingle-Cell Analysis0210 nano-technologySequence AnalysisAdultGanglionic eminenceAdolescentScienceNeurogenesisInhibitory postsynaptic potentialAmygdalaArticleGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesYoung AdultFetusmedicineHumansPreschoolProgenitorAgedCell NucleusSequence Analysis RNAInfant NewbornNeurosciencesInfantGeneral ChemistryAdolescent DevelopmentStem Cell ResearchNewborn030104 developmental biologynervous systembiology.proteinNeuronal developmentRNAlcsh:QTBR1Neuroscience
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Targeting brain and peripheral plasticity of the lipidome in acute kainic acid-induced epileptic seizures in mice via quantitative mass spectrometry.

2017

Epilepsy is a highly common chronic neurological disorder, manifested in many different types, affecting ~1% of the worldwide human population. The molecular mechanisms of epileptogenesis have not yet been clarified, and pharmacoresistance exhibited by 30-40% of epilepsy patients remains a major obstacle in medical care. Growing evidence indicates a role of lipid signalling pathways in epileptogenesis, thus lipid signals emerge as potential biomarkers for the onset and evolving course of the epileptic disorder, as well as potential therapeutic agents and targets. For this purpose, we applied a lipidomic strategy to unravel lipid alterations in brain regions, periphery tissues and plasma tha…

0301 basic medicineMaleKainic acidPopulationPharmacologyBiologyEpileptogenesisMass Spectrometry03 medical and health sciencesEpilepsychemistry.chemical_compoundOleoylethanolamideMice0302 clinical medicinemedicineAnimalseducationMolecular BiologyLungPalmitoylethanolamideeducation.field_of_studyEpilepsyKainic AcidNeuronal PlasticityFatty AcidsBrainHeartCell BiologyAnandamidemedicine.diseaseLipidsMice Inbred C57BL030104 developmental biologychemistrylipids (amino acids peptides and proteins)Epileptic seizuremedicine.symptom030217 neurology & neurosurgerySignal TransductionBiochimica et biophysica acta. Molecular and cell biology of lipids
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Functional Improvement after Photothrombotic Stroke in Rats Is Associated with Different Patterns of Dendritic Plasticity after G-CSF Treatment and G…

2016

We have previously shown that granulocyte-colony stimulating factor (G-CSF) treatment alone, or in combination with constraint movement therapy (CIMT) either sequentially or concomitantly, results in significantly improved sensorimotor recovery after photothrombotic stroke in rats in comparison to untreated control animals. CIMT alone did not result in any significant differences compared to the control group (Diederich et al., Stroke, 2012;43:185-192). Using a subset of rat brains from this former experiment the present study was designed to evaluate whether dendritic plasticity would parallel improved functional outcomes. Five treatment groups were analyzed (n = 6 each) (i) ischemic contr…

0301 basic medicineMalemedicine.medical_specialtyLightmedicine.medical_treatmentMovement10208 Institute of NeuropathologyIschemialcsh:Medicine610 Medicine & health1100 General Agricultural and Biological Sciences03 medical and health sciences0302 clinical medicine1300 General Biochemistry Genetics and Molecular BiologyInternal medicineNeuroplasticityGranulocyte Colony-Stimulating FactormedicineAnimalscardiovascular diseasesRats Wistarlcsh:ScienceSalineStrokePhysical Therapy Modalities1000 MultidisciplinaryMultidisciplinaryNeuronal Plasticitybusiness.industryPyramidal Cellslcsh:RDendritesRecovery of Functionmedicine.diseaseCombined Modality TherapyCortex (botany)SurgeryGranulocyte colony-stimulating factorConstraint-induced movement therapyStroke030104 developmental biologyEndocrinologyConcomitant570 Life sciences; biologylcsh:Qbusiness030217 neurology & neurosurgeryResearch ArticlePLoS ONE
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Formin 2 links neuropsychiatric phenotypes at young age to an increased risk for dementia

2017

Age-associated memory decline is due to variable combinations of genetic and environmental risk factors. How these risk factors interact to drive disease onset is currently unknown. Here we begin to elucidate the mechanisms by which post-traumatic stress disorder (PTSD) at a young age contributes to an increased risk to develop dementia at old age. We show that the actin nucleator Formin 2 (Fmn2) is deregulated in PTSD and in Alzheimer's disease (AD) patients. Young mice lacking the Fmn2 gene exhibit PTSD-like phenotypes and corresponding impairments of synaptic plasticity, while the consolidation of new memories is unaffected. However, Fmn2 mutant mice develop accelerated age-associated me…

0301 basic medicineMalememoriaAginggenetics [Stress Disorders Post-Traumatic]Diseasegenetics [Neuronal Plasticity]BioinformaticsdemenciaStress Disorders Post-TraumaticMice0302 clinical medicineRisk FactorsNews & ViewsAge of OnsetMice KnockoutNeuronal PlasticitybiologyGeneral NeuroscienceMicrofilament ProteinsNuclear Proteinsgenetics [Nuclear Proteins]FearadultoMiddle AgedAlzheimer's diseasephysiology [Aging]Phenotype3. Good healthPhenotypemiedoFormin 2Forminsgenetics [Aging]estres postraumaticoepidemiology [Stress Disorders Post-Traumatic]AdultHDAC inhibidorpsychology [Dementia]alzheimerForminsNerve Tissue Proteinsepidemiology [Dementia]Affect (psychology)General Biochemistry Genetics and Molecular Biology03 medical and health sciencesHDAC inhibitorMemorygenetics [Dementia]ddc:570medicineDementiaAnimalsHumansenvejecimientoMolecular Biologyphysiology [Memory]General Immunology and MicrobiologyPost-traumatic stress disordermedicine.diseaseYoung age030104 developmental biologyformin 2 protein mouseCase-Control StudiesSynaptic plasticitybiology.proteinDementiagenetics [Microfilament Proteins]complications [Stress Disorders Post-Traumatic]030217 neurology & neurosurgeryHomeostasis
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